The End of Alzheimer's: The First Program to Prevent and Reverse Cognitive Decline

In this paradigm shifting book, Dale Bredesen, MD, offers real hope to anyone goin to prevent and even reverse Alzheimer 's Disease and cognitive decline. Revealing that AD is not one condition, as it is currently treated, but three, nd of Alzheimer 's outlines 36 metabolic factors ( micronutrients, hormone levels, sleep) that can trigger " downsizing " in the brain. The protocol shows us how to rebalance these factors using lifestyle modifications like taking B12, eliminating gluten, or improving oral hygiene.

he results are remarkabl. Of the nex ten patients on the protocol, nine displayed significant improvement with 3-6 months; since then the protocol has yielded similar results with hundreds more. Now, nd of Alzheimer 's brings new hope to a wide audience of patients, caregivers, physicians, and treatment centers with a poignan look inside the science and a complete step-by-step plan that fundamentally changes how we treat and even think about AD.

Year of the Publication
Available Languages
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Original Title of the Book
The End of Alzheimer's: The First Program to Prevent and Reverse Cognitive Decline
Publication Date
Published August 22nd 2017 by Avery Publishing Group

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I lready have a list of people who ant to ead my copy.

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I do doubt that adding all of these supplements as you eliminate some pretty good foods is helpful.I can almost excuse that everyone cheating on their diet- as determined by visiting health coaches- does not automatically rule out the connection between the protocol and their improvements, given that you coul ot ave an issue with all 36 factors.

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Here is th review from University of Georgetown Professor, Dr Robert Hedaya, MD: Dr. Bredesen has written the first comprehensive guide to preventing, halting, and reversing Alzheimer 's Disease.

/Alzheimer ’ s Disease Letter to the Editor Wall Street JournalTime for a Better Approach To Alzheimer ’ s TreatmentsAlzheimer ’ s research has failed because of adherence to a flawed model.April 16, 2017 12:32 p.m. ET ONE IMPORTANT THING THAT WAS LEFT OUT OF THE BOOK: Dr. Bredesen 's outstanding resume: http: // ...

Easton Center for Alzheimer 's Disease Research at UCLA, Los Angeles, CA, 2013-2015.Activities and Awards1970, 1993-1997 Athletic Letters, Caltech ( Football, Track) 1970-1974 Alfred P.

Plott Chair in Gerontology, UCLA1996 Child Neurology Society Lectureship1996 Lou and Eleanor Gehrig Lectureship, Columbia University ( given for the most outstanding work on amyotrophic lateral sclerosis within the previous year) 1997 United Way Combined Health Agencies Health Hero ( annual award for outstanding medical research in San Diego) 2000 Arthur Cherkin Award for Research in Neurodegenerative Disease, UCLA2005 Gilman-Barbour Distinguished Lecturer, University of Michigan2010 Editorial Board Member, JBC2011 Associate Editor, J Alzheimer 's Disease2013 Turken International Lecturer, Neurology Grand Rounds at UCLA2014 Steven DeArmond Lecture, American Academy of Neuropathologists, Portland, ORPublications ( selected peer-reviewed publications) • Butterfield DA, Galvan V, Lange MB, Tang H, Sowell RA, Spilman P, Fombonne J, Gorostiza O, Zhang J, Sultana R and Bredesen DE.

In vivo oxidative stress in brain of Alzheimer disease transgenic mice: Requirement for methionine 35 in amyloid beta- peptide of APP.

Many neuronal and behavioral impairments in transgenic mouse models of Alzheimer 's disease are independent of caspase cleavage of the amyloid precursor protein.

PMID: 20053918; PMCID: PMC3064502• Spilman P, Podlutskaya N, Hart MJ, Debnath J, Gorostiza O, Bredesen D Richardson A, Strong R and Galvan V.

Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer 's disease.

PMID: 20376313; PMCID: PMC2848616• Zhang J, Gorostiza OF, Tang H, Bredesen DE and Galvan V.

PMID: 20847422; PMCID: PMC3968071• Descamps, O, Zhang, Q, John V, Bredesen DE.

Differential expression and redox proteomics analyses of an Alzheimer disease transgenic mouse model: effects of the amyloid-beta peptide of amyloid precursor protein ( Xi).

PMID: 21691801; PMCID: PMC3246043• Zhang J, Rao RV, Spilman P, Mangada J, Xie L, Vitelli C, Gorostiza OF, Madden DT, Zeng X, Jin K, Hart MJ, Bredesen DE, Galvan V.

PMID: 22174832; PMCID: PMC3235131• Libeu CP, Poksay K., John V, Bredesen DE.

Structural and Functional Alterations in Amyloid-B Precursor Protein Induced by Amyloid-B Peptides, J Alzheimers Dis 2011; 25:547-566.

Cellular effects of APOE4: Implications for Alzheimer 's disease.

PMID: 3175831; PMCID: PMC3736741• Rao R, Descamps O, John V, Bredesen DE.

PMID: 24194802; PMCID: PMC3806584• Sultana R, Robinson RA, Lange MB, Fiorini A, Galvan V, Fombonne J, Baker A, Gorostiza O, Zhang J, Cai J, Pierce WM, Bredesen DE, Butterfield DA.

Do proteomics analyses provide insights into reduced oxidative stress in the brain of an Alzheimer disease transgenic mouse model with an M631L amyloid precursor protein substitution and thereby the importance of amyloid-beta-resident methionine 35 in Alzheimer disease pathogenesis?

PMID: 21691801; PMCID: PMC3246043• Bredesen DE, John V.

PMID: 23703924; PMCID: PMC3779441• Descamps O, Spilman P, Zhang Q, Libeu CP, Poksay K, Gorostiza O, Campagna J, Jagodzinska B, Bredesen DE, John V.

PMID: 23948888; PMCID: PMC3971881• Zhang J, Spilman P, Chen S, Gorostiza O, Matalis A, Niazi K, Bredesen DE, Rao RV.

PMID: 23022695; PMCID: PMC3562756• Theendakara V, Patent A, Peters-Libeu C, Philpot B, Flores S, Descamps O, Poksay K, Zhang Q, Cailing G, Hart M, John V, Rao R, Bredesen D.

PMID: 24145446; PMCID: PMC3831497• Spilman P, Descamps O, Gorostiza O, Peters-Libeu C, Poksay K, Matalis A, Patent A, Rao R, John V, Bredesen D.

PMID: 24389031; PMCID: PMC4001856 [ Available on 2015/3/10 ] • Zhang Q, Descamps O, Hart MJ, Poksay K, Spilman P, Kane D, Gorostiza O, John V, Bredesen DE.

Paradoxical Effect of TrkA Inhibition in Alzheimer 's Disease Models.

PMID: 25324467; PMCID: PMC4221920• Zhang Q, Du G, John V, Kapahi P, Bredesen DE.

Journal of Alzheimer 's Disease, 2014 May.

( 2010) BACE, APP Processing, and Signal Transduction in Alzheimer 's Disease.

Role of Programmed Cell Death in Neurodegenerative Disease.

rated it

Another friend brought this book to me and reminded me to ead it and then to help him incorporate the protocol into his life to fend off mild cognitive impairment symptoms.

There are th few drugs that help a little bit, but fortunately, this type of one drug solution so common in medicine wo n't work because there are ozens of potential causes of the isease.

Patching one or two holes wo n't do the trick.The three main categories of the auses of the disease are: 1.

Metabolic issues, also caused by a myriad of factors, insulin resistance, lack of the vitamins needed to properly break down foods, unhealthy foods, lack of exercise, daylight, etc.

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I urge you to read anothe nove and ake the protocol seriously, it an change you and your loved ones lives.

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